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As an outlook, the simultaneous inhibition of essential oxidative stress response enzymes by the 5-nitroimidazole ether analogues causes a perfect storm of damage and attenuated rescue (Fig

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For example, early postnatal alcohol exposure leads to persistent hypermethylation of the FKBP5 promoter in the hippocampus, likely disrupting HPA axis feedback regulation and enhancing susceptibility to depression-like phenotypes through epigenetically mediated stress-response dysregulation 172

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