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Currently, there are two main strategies for targeting copper in cancer treatment

In the hypoxic environment, activation of the HIF-1/BNIP3 pathway induced BNIP3-associated mitophagy

This shifting metabolic state is why blanket antioxidant supplementation without baseline assessment may not align with individual needs and could theoretically interfere with adaptive cellular responses to the medication

Studies have shown that FAM134B deficiency renders cells more susceptible to apoptosis and is associated with hereditary sensory and autonomic neuropathy type IIB (HSAN IIB) [28]