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Sequential versus simultaneous use of chemotherapy and gonadotropin-releasing hormone agonist (GnRHa) among estrogen receptor (ER)-positive premenopausal breast cancer patients: effects on ovarian function, disease-free survival, and overall survival

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One plausible explanation is that fatty acids of various carbon chain lengths may regulate the conformational changes and transcriptional activities of PPARs in different or even opposing manners [26]

Due to HPG axis disruption, phthalates alter the levels of GnRH by interacting with genes of G protein-coupled receptors [GPCRs] on pituitary cells and disrupt FSH and LH ratio by interfering with their receptors on Leydig cells, which consequently disrupt the normal activity of steroidogenic enzymes and steroid hormones (142)