hypothesis glutathione persister cells

hypothesis glutathione persister cells Drug-tolerant in cancer: the cutting edges and future directions Frontiers | Exogenous cystine increases

Name: hypothesis glutathione persister cells Drug-tolerant in cancer: the cutting edges and future directions Frontiers | Exogenous cystine increases
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Frontiers Exogenous cystine increases susceptibility of drug resistant Salmonella to gentamicin by promoting oxidation of glutathione metabolism and imbalance of intracellular redox levels Glutathione peroxidase 4dependent glutathione highconsumption drives acquired platinum chemoresistance in lung cancerderived brain metastasis Liu 2021 Clinical and Translational Medicine Wiley Online Library JCI CD8+ T cells sustain antitumor response by mediating crosstalk between adenosine A2A receptor and glutathione GPX4 Frontiers Cysteine Depletion, a Key Action to Challenge Cancer Cells to Ferroptotic Cell Death

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Retrieved July 18, 2009

& Cambron, S

Selective modulation of glutathione levels in human normal versus tumor cells and subsequent differential response to chemotherapy drugs

have revealed that ferroptosis regulates PD-L1 expression in HNSCC through a dual mechanism: on the one hand, PD-L1 is upregulated via the ROS-driven NF-B signaling pathway (a membrane damage-independent pathway), and on the other hand, immune checkpoint molecule expression is promoted through ferroptosis stress-induced calcium influx (a membrane damage-dependent pathway)