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Wu et al (318) explored the relationship between miRNAs and oxidative stress-related genes, and found that cataract-regulated miRNAs could promote cataract formation not only by targeting the 3' UTR, which is a classic regulatory pathway, but also by binding to the TATA-box region of oxidative stress-related genes, leading to the subsequent elevation of pro-oxidative genes and inhibition of anti-oxidative genes