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Supporting this hypothesis, several studies have emphasized the involvement of oxidative stress in heavy metal toxicity

NO was originally identified as endothelium-derived relaxant factor based on the observations of Furchgott and Zawadzki [3]

CoQ10, a ROS scavenger and anti-inflammatory substance, has been shown to regulate Th17 and IL-17 through the STAT3 pathway, thereby inhibiting ferroptosis and ameliorating joint inflammation in CIA mice (124)

This alteration of the endothelium-mediated vasorelaxation is also observed in healthy subjects affected with moderate hyperhomocysteinemia (Tawakol et al, 1997