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In the human reference genome, there are seven GST gene families, each likely formed by gene duplications, and thus similar to each other in sequence and chromosomal location [18]

Extrinsic apoptotic pathways are activated by interactions between cell surface exposed death-inducing ligands such as FAS ligands (FASL) and tumor necrosis factor (TNF), and their cognate receptors Fas cell surface death receptor (FAS) and TNF receptor (TNFR) [110, 111]

A more credible translational narrative for PF in DPN will therefore depend on (i) clarifying the active molecular entity and DPN-contextualized exposure, (ii) improving methodological rigor and model relevance, and (iii) linking exposure to disease-modifying structural and functional outcomes using clinically meaningful measures

10.1126/scitranslmed.3006055 Sci