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Ferroptosis is increasingly implicated in a host of diseases, and unlike other cell death programs the physiological initiation of ferroptosis is conceived to occur not by an endogenous executioner, but by the withdrawal of cellular guardians that otherwise constantly oppose ferroptosis induction

Compared to the control group, those using Polyphenon E had reduced levels of the vascular endothelial growth factor (VEGF) from 24 months and significantly lower levels of serum hepatic growth factor (HGF) at 2 months

Using PbSOD1 and PbSOD3 knockdown strains we showed these genes are involved in the response of the fungus against host effector cells, particularly the oxidative stress response, and in a mouse model of infection