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GHK appears to directly target myofibroblasts, converting pathological persistent collagen deposition to physiological collagen contraction through ITG-1 activation and apoptotic elimination of excess myofibroblasts (Figure 5)

[60] Echeverria D, Aposhian HV, Woods JS, Heyer NJ, Aposhian MM, Bittner AC, Mahurin RK, Cianciola M

BrdU is considered a thymidine analog that integrates into the DNA of proliferating cells during the S-phase of the cell cycle, serving as a marker for cell division

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