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Description

Genetic variants that impact the carboxyl-terminal domain of FTL and modify its iron-binding capacity have been linked to the disease hereditary neuroferritinopathy, it is characterized by progressive neurologic symptoms and iron accumulation, particularly in the basal ganglia

The iron chelator DFO retards photoreceptor degeneration in animal models of RP, supporting a role for ferroptosis in RP progression (244)

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