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[7] Because it engages GIP and GLP-1 within one molecule, it is mechanistically distinct from CagriSema, which recruits amylin rather than GIP

Bao gi: Hp 30 vin

Dig Dis Sci 2010

Epidemiological data further support this connection: patients with inflammatory bowel disease exhibit a 28%30% increased risk of developing PD (Zhu et al., 2019), and Epstein-Barr virus infection has been linked to PD-like symptoms, highlighting the potential role of immune dysregulation in PD pathogenesis (Rajan et al., 2023