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Data was self-reported by 1,412 surveyed customers who checked in with their provider on their second check-in between 4/1/23 and 4/1/24

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In vitro, CD36, FATP, CPT1a, and ACOX1 were significantly downregulated in HK-2 cells treated with COM, recovered after PRMT1 knockdown, and further decreased after PRMT1 overexpression (Fig

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