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O.et al (2019)

reported that uptake and cytotoxicity of oxaliplatin increased in HEK293 cells overexpressing rat OCTN1, rat OCTN2, human OCTN1, and human OCTN2, and that OCTN1-mediated transport of oxaliplatin seemed to make a greater contribution to its neuronal accumulation and neurotoxicity compared to OCTN2 or OCTs (Jong et al., 2011)

Regulatory agencies are more likely to swiftly move forward with therapies that demonstrate consistent and reliable safety data

Inhibition of these enzymes by xenobiotics, including drugs and botanical products (precipitants), can lead to increased systemic exposure to the object drug and potential adverse or toxic effects