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We hypothesized that the observed lower GSH:GSSG ratio in asthmatic lung fluids and cells is at least partially driven by high oxidation of GSH and its export in response to greater intracellular 15LO1/PEBP1 pathway activity

The vitality of mitochondria extends across various physiological systems, emphasizing the necessity of an intact and fully functional mitochondrial architecture for efficient energy production [21]

Since the ER is a major base for lipid synthesis and has the largest membrane structure in the cell, it is speculated that the conversion of unsaturated lipids to lipid peroxides during ferroptosis may lead to changes in ER viscosity

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