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In PD, dopaminergic information transmitted by dopaminergic neurons decreases, while glutamatergic signaling in the basal ganglia increases and stimulates the release of dopamine through surviving dopaminergic neurons in the SNpc as a compensatory mechanism

Biomechanical modeling and 3D reconstruction studies points at the viscoelasticity of ONH structures as a determinant of the dynamic ONH response to changes in IOP