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The distinct actions of Myo-Ins and Mdivi-1 indicate that the Myo-Insdependent potentiation of ATP-mediated Ca signalling does not primarily depend on mitochondrial fission inhibition, consistent with a mechanism acting at the level of PIP metabolism or IP-mediated signalling, rather than through changes in mitochondrial structure

Sekhar RV, Patel SG, Guthikonda AP, Reid M, Balasubramanyam A, Taffet GE, Jahoor F

Conversely, when transsulfuration is stressed, methylation capacity may also be affected

The regulatory effect of NRF2 on those genes was well documented through antioxidative response element binding in the promoter or enhancers of target genes