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Description

At last, damaged mitochondria-released Cytochrome C triggers apoptotic processes by activating caspases (McGovern and Barreto, 2021) propagation in the long term (Trushina and McMurray, 2007

PD-1 can upregulate the expression of PD-L1 in tumor cells and ultimately inhibit the proliferation of T cells and reduce immune function by binding to its ligand PD-L1, thereby facilitating tumor cell immune escape [61,62,63,64], this is the mechanism by which tumor cells evade immune cells with the help of PD-1 in the absence of glutamine