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Evidence for a common non-heme chelatable-iron-dependent activation mechanism for semisynthetic and synthetic endoperoxide antimalarial drugs

doi: 10.3389/fpsyt.2021.682056 201 WangRHeMQuFZhangJXuJ

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Researchers have used GHK-Cu in cell-based assays to evaluate changes in gene expression, enzyme activation, and signaling protein phosphorylation associated with copper-dependent pathways[5]