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Heat stress induced apoptosis is triggered by transcription-independent p53, Ca2+ dyshomeostasis and the subsequent Bax mitochondrial translocation

Because Fe-S clusters function as essential cofactors for components of the electron transport chain, TCA cycle enzymes, and genome maintenance machinery, their stability depends on a reducing intracellular environment that is maintained, in part, by GSH 203,208

Additionally, the gut microbiota-derived metabolite capsiate relieves ferroptosis-related osteoarthritis through the regulation of solute carrier family 2 member 1 and HIF-1 [99]

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