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The brain has a high lipid content, which is the main target for oxidative damage

The authors show that the drastic growth arrest of the sir1 mutant is partially rescued when glutathione deficiency is introduced via the cad2 mutation, which decreases glutathione levels to 30% to 40% of wild-type levels (Cobbett et al., 1998), or pharmacologically through treatment with the GCL inhibitor buthionine sulfoximine

This synthetic phosphopeptide reached Phase III clinical trials, making it one of the few peptide-based therapies to advance that far in autoimmune disease research

[PubMed] [Google Scholar] 59.Jollow D.J., Mitchell J.R., Potter W.Z., Davis D.C., Gillette J.R., Brodie B.B