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Furthermore, evidence has emerged demonstrating that LINC01764 exhibits a specific binding affinity for hnRNPK, thereby promoting its interaction with the oncogene c-MYC and facilitating the translation of ribosomes within c-MYC

Results obtained from randomized clinical trials, however, were inconsistent and in some circumstances have indicated that antioxidants increase cancer risk (Klein et al., 2011

m , Venn diagram of differentially expressed genes in response to treatments

Mechanistic studies reveal that inflamed intestinal tissues in IBD patients and corresponding animal models display characteristic ferroptosis features, including GSH depletion, suppression of GPX4 activity, and abnormal iron deposition