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Possibly, the neuroprotective effect of Semax on ischemia-damaged nervous tissues includes the impact of Ca 2+ penetration into the cell on the regulatory processes

In healthy individuals, the v3 integrin facilitates endothelial cell migration and neovascularization, while p53 typically functions as an anti-angiogenic agent, inhibiting excessive angiogenesis

Interventions in this direction mainly manifest in two complementary logics: one is to indirectly block hypoxia-induced pro-oxidant signaling axes by inhibiting HIF function [486]