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Manipulation of diacylglycerol and ERK-mediated signaling differentially controls CD8+ T cell responses during chronic viral infection

In schizophrenia, immune activation upregulates pro-inflammatory cytokines (e.g., IFN-, IL-1, TNF-), which activate indoleamine 2,3-dioxygenase (IDO) via both IFN- receptor (IFN-R)-dependent and independent pathways (e.g., synergistic action of TLR4, IL-1R, TNF-R) (215)

4a, the G/D-CuP group exhibited the most intense CD206-associated red fluorescence signal among all treatment groups, indicating that G/D-CuP promotes macrophage polarization toward the M2 phenotype