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Philip Kistler Stroke Research Center, Department of Neurology, Massachusetts General Hospital, Harvard Medical School, Boston, USA, Boston, United States, 2 Center for Imaging Sciences and Medical Physics, Department of Medical Imaging, Hematology, and Clinical Oncology, Ribeirao Preto Medical School, University of So Paulo, So Paulo, Brazil, 3 Rush Alzheimer's Disease Center, Rush University Medical Center, Chicago, IL, USA, Chicago, United States, 4 Department of Biomedical Engineering, Illinois Institute of Technology, Chicago, IL, USA, Chicago, United States, 5 MassGeneral Institute for Neurodegenerative Disease, Massachusetts General Hospital, Harvard Medical School, Charlestown, MA, USA, Charlestown, United States Background and Aims: Cerebral Amyloid Angiopathy (CAA), characterized by amyloid- accumulation in leptomeningeal and cortical vessels, is a leading cause of intracerebral hemorrhage (ICH) lacking effective treatment options

However, as their role in the psychopathology of AIS is uncertain, the development of neuroprotective agents against them is currently not a priority

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Like glutathione, carnosine levels decrease with age (Bellia 2009