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DSIP, if effective, would layer on deeper slow-wave support rather than replace these staples

Rather than overriding natural processes like many pharmaceuticals, BPC-157 acts as a cytoprotective mediator, coordinating repair through targeted pathways: Angiogenesis (Vascular Recruitment): One of its most potent effects is the promotion of collateralization, the creation of new blood vessels

In preclinical models, BPC-157 has been associated with: Reduced inflammatory cytokine expression Improved transition from inflammatory phase to proliferative phase This creates an environment where fibroblasts can function more efficiently

The study also evaluated drug-induced rotation asymmetry, a measure of neurological deficit after unilateral MCAO