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Interestingly, Tex functionality can be restored with metabolic manipulation, as described above, through PGC-1 overexpression (121, 147), further supporting the notion that ability to maintain OXPHOS and FAO metabolism are necessary to avoid Tex phenotype

It is also very necessary to identify if TMAO contributes to chondrocytes injury through modulating the classical pathways of ferroptosis such as ACSL4/GPX4 or other specific pathways, such as those of iron-related proteins, lipid peroxidation products, and other sensitivities to mechanical stimulation

Its an emulsifier that also adds a little extra moisture and makes the texture feel luxurious

g Mitochondria/nuclear DNA ratio in gWAT from control and NDI1 mice after 10 weeks of HFD feeding (n = 6)