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There exists a dearth of clinical evidence on how dietary intake can aid in the prevention and control of cancer progression for an individual subject, but potential dietary interventions have been evaluated recently [128, 129]

Cholesterol-derived oxysterols induce structural and functional disruptions in key TJ components, including ZO-1, occludin, and junctional adhesion molecule-A (JAM-A), resulting in a weakened epithelial barrier that facilitates bacterial translocation and systemic inflammation [11, 83]

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This action is similar to natural GHRH, but with less effect on other hormones such as cortisol and prolactin