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JAK-STAT pathway alterations impair immune homeostasis by affecting immune cell development and function

However, during inflammation, these processes are impaired, and lipoprotein oxidation is intense and allows for their accumulation in the subendothelial space

this effect can be reversed by sulforaphane, a molecule with chemopreventive ability, and, in fact, represents a potential mechanism of action for sulforaphane-mediated induction of NRF2 [235]