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Activation of integrin-linked kinase is a critical prosurvival pathway induced in leukemic cells by bone marrow-derived stromal cells

The hyper-oxidation of 2-Cys Prx, in particular of Prx1, adds further chaperone function to these Prxs, but depends on certain motif elements downstream of the peroxidatic cysteine residue (GGLG and YF motifs) [23, 61]

This effect is further compounded by the inactivation of glutathione peroxidase 4 (GPX4) as ONOO can modify the selenocysteine residue within its active site, thereby disabling the only enzyme capable of directly reducing complex lipid hydroperoxides in biological membranes

Neurological Disorders Alzheimer's Disease (AD): In AD, alterations in PC metabolism contribute to synaptic dysfunction and neurodegeneration