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Oxidative stress induces MPT by targeting specific cysteine residues of cyclophillin D, which results in mitochondrial depolarization, oxidative phosphorylation uncoupling, release of intra-mitochondrial ions and metabolic intermediates, mitochondrial swelling and decrease in ATP synthesis

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The pathophysiology of hyperbilirubinemia in GS involves disruptions in bilirubin production, hepatic uptake, and glucuronidation, and transport, all of which are influenced by genetic factors

It has the ability to reduce neuro-inflammation which helps relieve the cognitive symptoms of conditions such as Lyme, Alzheimers disease, Parkinsons disease, and even post-concussion syndrome