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Hum Mol Genet 2004;13:70314

10.1161/STROKEAHA.121.036800 364 PengY.LuJ.FanL.DongW.JiangM

The position of GSH in the model is supported by data showing that mutation of two of the arginines, R73 and R74, results in complete loss of substrate saturation with GSH (with CDNB as the electrophilic second substrate)

Furthermore, studies reviewing the relationship between UVR and human biology are hindered by the infeasibility of measuring extended UVR exposure of populations