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In sharp contrast, modulation of H 2 S synthesis stimulates eNOS activities and NO production in the heart tissues and aortic rings, thus recovering endothelium-dependent relaxation and arterial elastance, as well as cardiac dysfunction in diabetic rats ( 2 S and NO levels in rats, and moxonidine treatment abrogated cardiovascular dysfunction in diabetic rats through restoring cardiac H 2 S and NO levels ( 2 S confers a protective effect on the myocardium in diabetic animals, which may be associated with the regulations of iNOS, eNOS, and NO production