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(57) analyzed ferroptosis-related gene expression patterns specifically in myocardium following coronary occlusion, revealing an immune-ferroptosis axis that influences post-infarction remodeling

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After 12 days, tumor in the BSO@MOF-LR group showed suppressed tumor growth compared with that in the free OXA and OXA@MOF-LR groups, owing to iron-based ROS production and BSO-mediated GSH inhibition in the tumor tissue, eventually resulting in BSO@MOF-LR-induced ferroptosis

As a functional measure of endothelial health, even modest increases in flow-mediated dilation (FMD) are associated with a lower risk of future cardiovascular events