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Integrating multi-omics approaches with functional validation in human tissues will be essential to translate ferroptosis-based interventions into clinically relevant therapies

This translocation of gut luminal pathobionts and products triggers inflammatory responses in the liver, involving resident hepatic macrophages, Kupffer cells, and other infiltrating immune cells that activates hepatic stellate cells (HSC), causing aberrant production of extracellular matrix (ECM) proteins and fibrosis development

It reduced memory impairment and increased malondialdehyde levels, glutathione, and SOD activity in the brain

It is important to note that while our LLM-driven approach provides valuable insights and efficiency in candidate gene prioritization, it does not replace the need for experimental validation