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To assess for possible kidney damage, we quantified cystatin-C instead of the typical markers such as blood urea nitrogen and creatinine ratio, as it is a better marker of renal homeostasis [46]

These data suggest that alternative oxidase can be crucial under nitrosative stress: The high alternative oxidase activity can maintain respiration at low cytochrome c oxidase activity and reduces the activity and hence superoxide production of cytochrome bc1 complex

54,55 Opa1 is localized in the inner mitochondrial membrane, which is responsible for the fusion of the inner membrane, and maintains the membrane structure

J Clin Immunol 2004