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Non-systemic intestine-targeted drugs

Sulforaphane exerts its anti-inflammatory effect against amyloid- peptide via STAT-1 dephosphorylation and activation of Nrf2/HO-1 cascade in human THP-1 macrophages

Remarkably, NAC also blocked microbiome changes along with systemic autoimmunity in lupus-prone MLR/lpr mice [18] , suggesting that gut dysbiosis is driven by oxidative stress on the organismal levels (Table 1)

Activated NF-B not only promotes the formation of OCs but also promotes the production of proinflammatory factors, forming a vicious cycle of positive feedback regulation, which further increases the progression of RA