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Heliyon 7 , e07605 (2021)

This transcriptional regulation complements the previously described suppression of pro-inflammatory mediators, such as NO, PGE2, and ROS, and the inhibition of NF-B and JNK signaling pathways

Baseline demographic and clinical data, including age, gender, and laboratory markers, were collected

This protective effect is further supported by observations that GPX4 levels are diminished in cardiomyocytes of patients with diabetic heart disease [219], suggesting therapies of ferroptosis antioxidants have shown promise in alleviating diabetes-induced oxidative stress and cardiac dysfunction