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As a relatively stable and low-inflammatory precursor phase, AMSU tends to induce heterogeneous nucleation and deposition on damaged articular cartilage surfaces, synovial connective tissue, or cell-derived vesicles at various biological interfaces, and may participate in the uric acid crystallization kinetics process within a certain timescale (3, 1014)

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It is recognized that GSH controls the activity of thiol-dependent proteins by keeping the SH groups of protein in a reduced state and preventing them from oxidation [75]

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