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It has been found that the ROS-Nrf2-antioxidant response element axis may be accountable for the elevation of SLC7A11, which is considered a potential compensatory mechanism to repress ferroptosis in HH

139 , 370372, doi: 10.1164/ajrccm/139.2.370 (1989)

These observations offer a rationale for studying 1-AR antagonists to prevent CSS and its appalling consequences in people who are at risk for developing severe COVID-19