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Our research also verified that decreased expression of GCLM was responsible for the decreased activity of GCL, and that GCL activity was enhanced after the resumption of GCLM to promote GSH synthesis, which directly retarded ferroptosis

in-state residence was associated with 78% lower odds (aOR 0.22, 95% CI 0.05-0.77, p=0.03) of relapse within 1 year post-LT

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