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Bacterial vaginosis and health-associated bacteria modulate the immunometabolic landscape in 3D model of human cervix

Kojer et al., The redox couples present in organelles of the exocytic pathway display redox poises similar to the extracellular space

IL-1 promotes chondrocyte apoptosis and inhibits the synthesis of cartilage matrix components, whereas TNF- exacerbates joint inflammation and cartilage damage by stimulating the production of inflammatory mediators like nitric oxide and prostaglandin E2 [21, 22]

Therefore, macrophage ferroptosis may be considered to be involved in the progression of AS (97) and could serve as a potential target for intervention with AS progression