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Acute stress-induced liver damage can lead to infections or shock, posing a serious threat to health and life ( However, in addressing the critical challenge of stress-induced liver injury, traditional pharmacological treatments, while somewhat effective, often fail to fully meet clinical needs

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burgdorferi utilizes non-toxin adenylate cyclase cyaB , an enzyme that produces secondary messenger cyclic adenosine monophosphate (cAMP), to modify borrelial gene expression and protein production in order to enhance its virulence in the vertebrate host [109]

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