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For decades, the prevailing view has been that homotypic K48-linked polyubiquitin chains represent the canonical degradation signal recognized by the proteasome

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In support of the NRF2 pathway being a potential target for the treatment of ALS, studies have shown that NRF2 overexpression in astrocytes provided neuroprotection against mutated SOD1-induced toxicity and increased the median survival of mutant SOD1 transgenic mice (Vargas et al., 2008