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Importantly, the absence of substantial protection in experimental AP after genetic MCU ablation does not negate the pathogenic role of mitochondrial calcium overload, but rather indicates that MCU-dependent matrix Ca 2+ uptake is not the sole determinant of disease severity

Moreover, elevated levels of phthalates in pubertal boys and girls were associated with higher DNA methylation levels at the thyroid hormone receptor interactor 6 (TRIP6) promoter region, a gene associated with pubertal onset (87, 88)

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