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In this case, glutathione (GSH) is functioning as a storage mechanism for cysteine which is otherwise unstable in its free form

10.1016/j.cell.2005.06.022 Summary Keywords kinase, mitochondrial morphology, mitochondria, apoptosis, mitochondrial associated endoplasmic reticulum membranes (MAMs), reactive oxygen species (ROS), disease adaptation Citation Skalka GL, Tsakovska M and Murphy DJ (2024) Kinase signalling adaptation supports dysfunctional mitochondria in disease