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Analyses of macrophage viability in IBD are limited

Exosome-based delivery of natural products in cancer therapy

Furthermore, while ferroptosis induction has attracted major therapeutic interest, ferroptosis resistance should not be viewed as a passive absence of death signaling, but as an adaptive survival program selected during platinum exposure, PARP inhibition, hypoxia, and metastatic dissemination

PLs containing PUFAs are the primary substrates for lipid peroxidation in ferroptosis and are upregulated by enzymes such as acyl-CoA synthetase long-chain family member 4 (ACSL4), lysophosphatidylcholine acyltransferase 3 (LPCAT3), ALOXs, and Cytochrome P450 oxidoreductase(POR) (41).The membrane remodeling enzymes, ACSL4 (42) and LPCAT3, are key drivers of ferroptosis