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Neuregulins in neurodegenerative diseases

In support of this increased NK cytotoxic potential, the expression of the NK cytotoxic effector-associated gene perforin, as determined by flow-cytometric analysis or quantitative real-time PCR analysis in CD56 + NK cells, was also significantly increased in the above IGF-1-induced CD34 + -derived NK cells compared with the cells without IGF-1 (Fig

FIGURE 3 3.3 Neuroprotective activity Alzheimers disease (AD) is pathologically defined by the accumulation of amyloid-beta (A) plaques and hyperphosphorylated Tau proteins, disrupting the autophagy-lysosomal pathway (ALP) and causing neuronal death (Zheng et al., 2021) (Table 1, Row 18)

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