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Vitetta, L., Zhou, J., Manuel, R., Dal Forno, S., Hall, S., & Rutolo, D

Complement-dependent cytotoxicity involves the activation and deposition of complement components on the target cell surface, ultimately leading to the formation of the membrane attack complex (MAC)

Tivantinib (ARQ 197) is a staurosporine derivative and it represents the first non-ATP-competitive c-Met inhibitor that binds to the dephosphorylated c-Met kinase in vitro

It is also worth noting that a large cluster of long heparan polymers on the cell surface could be associated with multiple dimeric c-MET/HGF complexes and in principle induce the higher-order oligomerization of c-METs, and potentially lead to the formation of even larger biomolecular condensates on the cell membrane