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[DOI] [PMC free article] [PubMed] [Google Scholar] Yang SR, Valvo S, Yao H, Kode A, Rajendrasozhan S, Edirisinghe I, Caito S, Adenuga D, Henry R, Fromm G, Maggirwar S, Li JD, Bulger M, Rahman I

Gasparovic C, Song T, Devier D, Bockholt HJ, Caprihan A, Mullins PG, et al

On the other hand, FoB rely on OXPHOS to a lower extent: Gclc -deficient FoB lose CI and CII functionality, but these cells increase glycolysis to replenish their ATP pool to the extent that they persist in Gclc fl/fl Mb1 Cre + hosts

Rather than abandoning the association of Hcy with CVD, revisiting the interventional approaches and interpretation of data may likely help in a better understanding of the relevance of Hcy to CVD