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Tran CD, Miller LV, Krebs NF, Lei S, Hambidge KM (2004)

Expression of stress response genes is delayed in Gstp1/2 / mice after PH / ROS modulate MAPK activation such as JNK, which is essential for regulating the intracellular redox status and the balance between cell proliferation and cell death

26 Other possible mechanisms include improvements in mitochondrial function and a reduction of glutamate, an excitatory neurotransmitter, which is also a precursor to GSH